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PreviewIssue DateTitleContributor(s)
2005Telomere-mediated mitotic disturbances in immortalized ovarian epithelial cells reproduce chromosomal and breakpoints from ovarian carcinomaGisselsson, D; Lv, M; Tsao, SW; Man, C; Jin, C; Höglund, M; Yok, LK; Jin, Y
2004The candidate tumor suppressor gene BLU, located at the commonly deleted region 3p21.3, is an E2F-regulated, stress-responsive gene and inactivated by both epigenetic and genetic mechanisms in nasopharyngeal carcinomaSrivastava, G; Tsao, SW; Qiu, GH; Tan, LKS; Loh, KS; Lim, CY; Tsai, ST; Tao, Q
2000Effect of p16INK4a on chemosensitivity in nasopharyngeal carcinoma cells.Kwong, DL; Tsao, SW; Nicholls, JM; Chow, LS; Wang, X; Sham, JS
2000Structural changes and alteration in expression of TGF-β1 and its receptors in prostatic intraepithelial neoplasia (PIN) in the ventral prostate of noble ratsWong, YC; Xie, W; Tsao, SW
2000Sex hormone-induced mammary carcinogenesis in the female Noble rats: Expression of Bcl-2 and Bax in hormonal mammary carcinogenesisXie, B; Tsao, SW; Wong, YC
2002Id-1 stimulates serum independent prostate cancer cell proliferation through inactivation of p16INK4a/pRB pathwayOuyang, XS; Wang, X; Ling, MT; Wong, HL; Tsao, SW; Wong, YC
2008Proteomic comparison of nasopharyngeal cancer cell lines C666-1 and NP69 identifies down-regulation of annexin II and β 2-tubulin for nasopharyngeal carcinomaChan, CML; Wong, SCC; Lam, MYY; Hui, EP; Chan, JKC; Lo, ESF; Cheuk, W; Wong, MCK; Tsao, SW; Chan, ATC
2007Id-1 activation of PI3K/Akt/NFκB signaling pathway and its significance in promoting survival of esophageal cancer cellsLi, B; Tsao, SW; Ling, MT; Wong, YC; Cheung, ALM; Cheung, PY; Wang, X
2004Id-1-induced Raf/MEK pathway activation is essential for its protective role against taxol-induced apoptosis in nasopharyngeal carcinoma cellsCheung, HW; Ling, MT; Tsao, SW; Wong, YC; Wang, X
2007S-allylcysteine, a water-soluble garlic derivative, suppresses the growth of a human androgen-independent prostate cancer xenograft, CWR22R, under in vivo conditionsChu, Q; Lee, DTW; Tsao, SW; Wang, X; Wong, YC